THE ROLE OF ENDOGENOUS INTOXICATION IN THE REGULATION OF BCL-2 GENE EXPRESSION IN THE DYNAMICS OF EXPERIMENTAL SUBHEPATIC OBSTRUCTIVE JAUNDICE
Author:
Publisher
Grodno State Medical University
Subject
Pharmacology (medical)
Reference16 articles.
1. 1. Cherepanova MA. Osobennosti jekspressii antiapoptoticheskogo belka Bcl-2 v pecheni v modeli ozhirenija i saharnogo diabeta 2-go tipa i pri korrekcii linagliptinom [Peculiarities of the antiapoptotic Bcl-2 protein expression in liver in a model of obesity and type 2 diabetes and with linaglyptin correction]. Acta Biomedica Scientifica. 2018;3(1):116-119. doi: 10.29413/ABS.2018-3.1.18.(Russian).
2. 2. Komarceva IA, Popov JeN, Komarceva EV, Zaika AV. Promotor apoptoza gen r53 i antiapoptoznyj gen Bcl-2 v proliferirujushhih kletkah. Ukrainskij zhurnal klinichnoi ta laboratornoi medicini. 2009;4(2):162-167. (Russian).
3. 3. Yin XM, Oltvai ZN, Korsmeyer SJ. BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax. Nature. 1994;369(6478):321-333. doi: 10.1038/369321a0.
4. 4. Sato T, Hanada M, Bodrug S, Irie S, Iwama N, Boise LH, Thompson CB, Golemis E, Fong L, Wang HG, Reed JС. Interactions among members of the Bcl-2 protein family analyzed with a yeast two-hybrid system. Proc. Natl. Acad. Sci. 1994;91(20):9238-9242. doi: 10.1073/pnas.91.20.9238.
5. 5. Reed JC. Bcl-2 and the regulation of programmed cell death. J. Cell Biol. 1994;124(1-2):1-6. doi: 10.1083/jcb.124.1.1.
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