Involvement of Notch1 and ALK4/5 Signaling Pathways in Renal Tubular Cell Death: Their Application to Clarification of Cadmium Toxicity
Author:
Affiliation:
1. Department of Hygiene and Public Health, Tokyo Women’s Medical University
Publisher
Japanese Society for Hygiene
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jjh/75/0/75_20007/_pdf
Reference73 articles.
1. (1) Fujiki K, Inamura H, Matsuoka M. Detrimental effects of Notch1 signaling activated by cadmium in renal proximal tubular epithelial cells. Cell Death Dis 2014;5:e1378.
2. (2) Fujiki K, Inamura H, Sugaya T, Matsuoka M. Blockade of ALK4/5 signaling suppresses cadmium- and erastin-induced cell death in renal proximal tubular epithelial cells via distinct signaling mechanisms. Cell Death Differ 2019;26:2371–2385.
3. (4) Friberg L. Cadmium and the kidney. Environ Health Perspect 1984;54:1–11.
4. (5) Nordberg GF, Fowler BA, Nordberg M. Cadmium. In: Nordberg GF, Fowler BA, Nordberg M (eds). Handbook on the Toxicology of Metals. London: Academic Press, 2014, 667–716.
5. (6) Liu G, Zou H, Luo T, Long M, Bian J, Liu X, et al. Caspase-dependent and caspase-independent pathways are involved in cadmium-induced apoptosis in primary rat proximal tubular cell culture. PLoS One 2016;11:e0166823.
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