Changes in the activity of lysosomal cysteine proteases of plasma mononuclear and polymorphonuclear blood leukocytes in Alzheimer’s disease

Author:

Sorokina M. G.1ORCID,Fomina M. A.1ORCID,Petrov D. S.1ORCID,Korotkova N. V.1ORCID

Affiliation:

1. Ryazan State Medical University

Publisher

Siberian State Medical University

Subject

Molecular Medicine

Reference20 articles.

1. Gavrilova S. I. The pre-trial stage of Alzheimer’s disease: cUrrent approaches to diagnosis and pharmacotherapy. Neurology Psychiatry. 2017; 8 (137): 44–49 (in Russ.).

2. Bachinskaja N.Ju. Alzheimer’s disease. Zhurnal Nevrologii im. B.M. Man’kovskogo. 2013; 1: 88–102 (in Russ.).

3. Sokolik V.V. Alzheimer’s disease: genetic predisposition, biochemical mechanisms and mental manifestations. Ukrainskij Vestnik Psihonevrologii. 2007; 3(52): 101–105 (in Russ.).

4. Cermak S., Kosicek M., Mladenovic-Djordjevic A. Loss of cathepsin B and L leads to lysosomal dysfunction, NPC-like cholesterol sequestration and accumulation of the key Alzheimer’s proteins. PLOS ONE. 2016; 30: 1–17. DOI:10.1371/journal.pone.0167428.

5. Furman Ju.V., Smahtin M.Ju. Some functions of proteolytic enzymes in health and disease. Biologicheskie Nauki. 2017; 4 (13): 3–4 (in Russ.).

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