Effects of Agmatine on GABAAReceptor Antagonist-induced Tactile Allodynia
Author:
Affiliation:
1. Department of Anesthesiology and Pain Medicine, Anesthesia and Pain Research Institute, Yonsei University College of Medicine, Seoul, Korea.
2. Department of Neuroscience, Postgraduated Medical School of Yamaguchi University, Ube, Japan.
Publisher
Korean Pain Society
Subject
Anesthesiology and Pain Medicine
Link
https://synapse.koreamed.org/pdf/10.3344/kjp.2008.21.3.173
Reference34 articles.
1. Pathobiology of neuropathic pain
2. Physiopathologie de la douleur neuropathique : revue des modèles expérimentaux et des mécanismes proposés
3. Loss of glycinergic and GABAergic inhibition in chronic pain—contributions of inflammation and microglia
4. Behavioral and autonomic correlates of the tactile evoked allodynia produced by spinal glycine inhibition: effects of modulatory receptor systems and excitatory amino acid antagonists
5. The contribution of GABAA and glycine receptors to central sensitization: disinhibition and touch-evoked allodynia in the spinal cord
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1. Effect of intrathecal glycine and related amino acids on the allodynia and hyperalgesic action of strychnine or bicuculline in mice;Korean Journal of Anesthesiology;2010
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