KCa3.1 Inhibition of Macrophages Suppresses Inflammatory Response Leading to Endothelial Damage in a Cell Model of Kawasaki Disease-Reference-Cited by-同舟云学术

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KCa3.1 Inhibition of Macrophages Suppresses Inflammatory Response Leading to Endothelial Damage in a Cell Model of Kawasaki Disease

Published:2021-03 Issue: Volume:Volume 14 Page:719-735
ISSN:1178-7031
Container-title:Journal of Inflammation Research
language:en
Short-container-title:JIR

Author:

Zheng Fenglei,Tao Yijing,Liu Jingjing,Geng Zhimin,Wang Ying,Wang Yujia,Fu Songling,Wang Wei,Xie Chunhong,Zhang Yiying,Gong Fangqi

Publisher

Informa UK Limited

Subject

Immunology,Immunology and Allergy

Reference60 articles.

1. Diagnosis, Treatment, and Long-Term Management of Kawasaki Disease: A Scientific Statement for Health Professionals From the American Heart Association

2. A Single Intravenous Infusion of Gamma Globulin as Compared with Four Infusions in the Treatment of Acute Kawasaki Syndrome

3. Long-term Consequences of Kawasaki Disease

4. Epidemiologic Features of Kawasaki Disease in Japan: Results of the 2007–2008 Nationwide Survey

5. Gamma globulin re-treatment in Kawasaki disease

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1. KCa3.1 promotes the migration of macrophages from epicardial adipose tissue to induce vulnerability to atrial fibrillation during rapid pacing;Canadian Journal of Cardiology;2024-08

2. Neonatal Chlamydia muridarum respiratory infection causes neuroinflammation within the brainstem during the early postnatal period;Journal of Neuroinflammation;2024-06-15

3. Regulation of T Lymphocyte Functions through Calcium Signaling Modulation by Nootkatone;International Journal of Molecular Sciences;2024-05-11

4. Challenges in the Therapeutic Targeting of KCa Channels: From Basic Physiology to Clinical Applications;International Journal of Molecular Sciences;2024-03-04

5. M2 macrophage‑derived exosomes alleviate KCa3.1 channel expression in rapidly paced HL‑1 myocytes via the NF‑κB (p65)/STAT3 signaling pathway;Molecular Medicine Reports;2024-02-08

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