Mutation of MET D1228N as an Acquired Potential Mechanism of Crizotinib Resistance in NSCLC with MET Y1003H Mutation-Reference-Cited by-同舟云学术

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Mutation of MET D1228N as an Acquired Potential Mechanism of Crizotinib Resistance in NSCLC with MET Y1003H Mutation

Published:2024-08 Issue: Volume:Volume 15 Page:123-128
ISSN:1179-2728
Container-title:Lung Cancer: Targets and Therapy
language:en
Short-container-title:LCTT

Author:

Zhu Jinlian,Chen Jie,Liu Wei,Zhang Junling^ORCID,Gu Yulan

Publisher

Informa UK Limited

Reference22 articles.

1. Functional Analysis of MET Exon 14 Skipping Alteration in Cancer Invasion and Metastatic Dissemination

2. MET alterations in NSCLC—Current Perspectives and Future Challenges

3. Mechanisms of acquired resistance to first- and second-generation EGFR tyrosine kinase inhibitors

4. MET in Lung Cancer: Biomarker Selection Based on Scientific Rationale

5. Somatic Mutations Lead to an Oncogenic Deletion of Met in Lung Cancer

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