<p>Innate Immunity and Cell Surface Receptors in the Pathogenesis of COPD: Insights from Mouse Smoking Models</p>-Reference-Cited by-同舟云学术

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Innate Immunity and Cell Surface Receptors in the Pathogenesis of COPD: Insights from Mouse Smoking Models

Published:2020-05 Issue: Volume:Volume 15 Page:1143-1154
ISSN:1178-2005
Container-title:International Journal of Chronic Obstructive Pulmonary Disease
language:en
Short-container-title:COPD

Author:

De Cunto Giovanna^ORCID,Cavarra Eleonora^ORCID,Bartalesi Barbara^ORCID,Lucattelli Monica^ORCID,Lungarella Giuseppe^ORCID

Publisher

Informa UK Limited

Subject

Public Health, Environmental and Occupational Health,Health Policy,Pulmonary and Respiratory Medicine,General Medicine

Reference110 articles.

1. Rahman I, De Cunto G, Sundar IK, Lungarella G. Vulnerability and genetic susceptibility to cigarette smoke-induced emphysema in mice. Am J Respir Cell Mol Biol. 2017;57(3):270–271. doi:10.1165/rcmb.2017-0175ED.

2. Cigarette Smoke in Research

3. Neutrophil Elastase Contributes to Cigarette Smoke-Induced Emphysema in Mice

4. Different lung responses to cigarette smoke in two strains of mice sensitive to oxidants

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4. Anti-Inflammatory and Anti-Airway Remodeling Activities of Jakyakgamcho-Tang in a Mouse Model of COPD;Applied Sciences;2022-08-29

5. Smoking Cessation in Mice Does Not Switch off Persistent Lung Inflammation and Does Not Restore the Expression of HDAC2 and SIRT1;International Journal of Molecular Sciences;2022-08-14

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