<p>PTPLAD2 and USP49 Involved in the Pathogenesis of Smoke-Induced COPD by Integrative Bioinformatics Analysis</p>-Reference-Cited by-同舟云学术

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PTPLAD2 and USP49 Involved in the Pathogenesis of Smoke-Induced COPD by Integrative Bioinformatics Analysis

Published:2020-10 Issue: Volume:Volume 15 Page:2515-2526
ISSN:1178-2005
Container-title:International Journal of Chronic Obstructive Pulmonary Disease
language:en
Short-container-title:COPD

Author:

Zhang Qiang,Song Wei,Ayidaerhan Nahemuguli,He Zheng

Publisher

Informa UK Limited

Subject

Public Health, Environmental and Occupational Health,Health Policy,Pulmonary and Respiratory Medicine,General Medicine

Reference26 articles.

1. Geographic distribution of COPD prevalence in the world displayed by Geographic Information System maps

2. Chronic obstructive pulmonary disease

3. The Pathology of Chronic Obstructive Pulmonary Disease

4. Prevalence and risk factors of chronic obstructive pulmonary disease in China (the China Pulmonary Health [CPH] study): a national cross-sectional study

5. Network-based approach to prediction and population-based validation of in silico drug repurposing

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1. Identification and Validation of Aging Related Genes Signature in Chronic Obstructive Pulmonary Disease;COPD: Journal of Chronic Obstructive Pulmonary Disease;2024-08-13

2. Copper homeostasis dysregulation in respiratory diseases: a review of current knowledge;Frontiers in Physiology;2024-05-31

3. TET2 stabilized by deubiquitinase USP21 ameliorates cigarette smoke-induced apoptosis in airway epithelial cells;iScience;2024-03

4. The genomic landscape of chronic obstructive pulmonary disease: Insights from nutrigenomics;Clinical Nutrition ESPEN;2024-02

5. Bioinformatics analysis of PLA2G7 as an immune-related biomarker in COPD by promoting expansion and suppressive functions of MDSCs;International Immunopharmacology;2023-07

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