PRKAG2 and PRKAG3 genes in patients with Wolff-Parkinson-White syndrome: a literature review

Author:

Tolstokorova Yu. A.1ORCID,Nikulina S. Yu.1ORCID,Chernova A. A.2ORCID,Maksimov V. N.3ORCID,Makarov Ya. A.1

Affiliation:

1. V. F. Voino-Yasenetsky Krasnoyarsk State Medical University

2. V. F. Voino-Yasenetsky Krasnoyarsk State Medical University; Federal Siberian Research Cardiology Center

3. Research Institute of Therapy and Preventive Medicine — branch of the Institute of Cytology and Genetics

Abstract

Wolff-Parkinson-White syndrome (WPW) is a syndrome with early ventricular excitation due to the abnormal electrical conduction through an accessory atrioventricular pathway, and is usually accompanied by supraventricular tachycardia. There is a proven genetic component in the development of this syndrome. This review presents current literature data on the association of nucleotide sequence variants of the PRKAG2 and PRKAG3 genes in patients with WPW.

Publisher

Silicea - Poligraf, LLC

Subject

Cardiology and Cardiovascular Medicine

Reference31 articles.

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2. Kuzmin YuV, Bakalov SA. Diagnosis and treatment of WPW syndrome. Atmosphere. Cardiology News 2007;1:2-5. (In Russ.)

3. Saifudinov RG, Pak EV, Gilyadova AR, et al. Wolf-Parkinson-White syndrome in cardiologist practice. Bulletin of Modern Clinical Medicine. 2010;3(3):68-73. (In Russ.)

4. Ardashev AV, Rybachenko MS, Zhelyakov EG, et al. Wolf-Parkinson-White syndrome: classification, clinical manifestations, diagnosis and treatment. Cardiology. 2009;10:84- 94. (In Russ.)

5. Chernova АА, Nikulina SY, Matyushin GV, et al. Signs of connective tissue dysplasia and the type 3 endothelial nitrogen synthase (NOS3) gene in Wolf-Parkinson-White syndrome. Therapy. 2020;6:27-34. (In Russ.) doi:10.18565/therapy.2020.6.27-34

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