Association of polymorphic variants rs6684209 and rs7521023 of the calsequestrin gene (CASQ2) with contractile myocardial function in patients with coronary artery disease
Author:
Affiliation:
1. Research Institute of Cardiology, Tomsk National Research Medical Center
2. not
Publisher
Silicea - Poligraf, LLC
Subject
Cardiology and Cardiovascular Medicine
Reference12 articles.
1. Mareev VY, Fomin IV, Ageev FT, et al. Russian Heart Failure Society, Russian Society of Cardiology. Russian Scientific Medical Society of Internal Medicine Guidelines for Heart failure: chronic (CHF) and acute decompensated (ADHF). Diagnosis, prevention and treatment. Kardiologiia. 2018;58(6S):8-158. (In Russ.). doi:10.18087/cardio.2475.
2. Lehnart SE, Maier LS, Hasenfuss G. Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart. Heart Fail Rev. 2009;14:213-24. doi:101007/s10741-009-9146-x.
3. Rebrova TY, Muslimova EF, Kondratieva DS, et al. The role of Ca2+-ATPase 2A (ATP2A2), ryanodine receptors (RYR2), and calsequestrin (CASQ2) gene polymorphisms in the development of heart failure. Russian Journal of Genetics. 2018;54(6):604-8. doi:10.1134/S102279541806008X.
4. Glukhov AV, Kalyanasundaram A, Lou Q, et al. Calsequestrin 2 deletion causes sinoatrial node dysfunction and atrial arrhythmias associated with altered sarcoplasmic reticulum calcium cycling and degenerative fibrosis within themouse atrial pacemaker complex. European Heart Journal. 2015;36:686-97. doi:10.1093/eurheartj/eht452.
5. Estrada-AvileAs R, RodroAguez G, Zarain-Herzberg A. The cardiac calsequestrin gene transcription is modulated at the promoter by NFAT and MEF-2 transcription factors. PLoS. 2017;12(9):e0184724. doi:10.1371/journal.pone.0184724.
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