Growth hormone (GH)-transgenic insulin-like growth factor 1 (IGF1)-deficient mice allow dissociation of excess GH and IGF1 effects on glomerular and tubular growth-Reference-Cited by-同舟云学术

Growth hormone (GH)-transgenic insulin-like growth factor 1 (IGF1)-deficient mice allow dissociation of excess GH and IGF1 effects on glomerular and tubular growth

Published:2016-03 Issue:5 Volume:4 Page:e12709
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Blutke Andreas¹,Schneider Marlon R.²,Wolf Eckhard²,Wanke Rüdiger¹

Affiliation:

1. Institute of Veterinary Pathology at the Centre for Clinical Veterinary Medicine; Ludwig-Maximilians-University Muenchen; Munich Germany

2. Chair for Molecular Animal Breeding and Biotechnology; Gene Centre; Ludwig-Maximilians-University Muenchen; Munich Germany

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.12709/fullpdf

Reference65 articles.

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3. Genetic dissection of IGF1-dependent and -independent effects of permanent GH excess on postnatal growth and organ pathology of mice;Blutke;Mol. Cell. Endocrinol.,2014

4. Multiple consequences of human growth hormone expression in transgenic mice;Brem;Mol. Biol. Med.,1989

5. The role of the type I insulin-like growth factor receptor (IGF-IR) in glomerular integrity;Bridgewater;Growth Horm. IGF Res.,2008

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