Effect of NBCe1 deletion on renal citrate and 2-oxoglutarate handling

Author:

Osis Gunars1,Handlogten Mary E.1,Lee Hyun-Wook1,Hering-Smith Kathleen S.2,Huang Weitao2,Romero Michael F.3,Verlander Jill W.1,Weiner I. David14

Affiliation:

1. Division of Nephrology, Hypertension and Renal Transplantation; University of Florida College of Medicine; Gainesville Florida

2. Renal Division; Tulane University College of Medicine; New Orleans Louisiana

3. Department of Physiology & Biomedical Engineering and Nephrology & Hypertension; Mayo Clinic College Of Medicine; Rochester Minnesota

4. Nephrology and Hypertension Section; North Florida/South Georgia Veterans Health System; Gainesville Florida

Funder

National Institutes of Health

Department of Veterans Affairs

Publisher

Wiley

Subject

Physiology (medical),Physiology

Cited by 13 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. The proximal tubule through an NBCe1-dependent mechanism regulates collecting duct phenotypic and remodeling responses to acidosis;American Journal of Physiology-Renal Physiology;2023-01-01

2. Acid-base effects of combined renal deletion of NBCe1-A and NBCe1-B;American Journal of Physiology-Renal Physiology;2022-02-01

3. NBCe1-A is required for the renal ammonia and K+ response to hypokalemia;American Journal of Physiology-Renal Physiology;2020-02-01

4. Regulation of renal NaDC1 expression and citrate excretion by NBCe1-A;American Journal of Physiology-Renal Physiology;2019-08-01

5. Expression of the B splice variant of NBCe1 (SLC4A4) in the mouse kidney;American Journal of Physiology-Renal Physiology;2018-09-01

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