Contrasting effects of hyperoxia on GM-CSF gene transcription in alveolar epithelial cells and T cells
Author:
Affiliation:
1. Department of Veterans Affairs Medical Center; Salt Lake City Utah USA
2. Division of Respiratory; Critical Care and Occupational Pulmonary Medicine; University of Utah School of Medicine; Salt Lake City Utah USA
Funder
Department of Veterans Affairs
Publisher
Wiley
Subject
Physiology (medical),Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.12324/fullpdf
Reference43 articles.
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2. Post-transcriptional regulation of granulocyte-macrophage colony-stimulating factor synthesis in murine T cells;Bickel;J. Immunol.,1990
3. Granulocyte/macrophage-colony-stimulating factor (GM-CSF) regulates lung innate immunity to lipopolysaccharide through Akt/Erk activation of NFkappa B and AP-1 in vivo;Bozinovski;J. Biol. Chem.,2002
4. Innate immune responses to LPS in mouse lung are suppressed and reversed by neutralization of GM-CSF via repression of TLR-4;Bozinovski;Am. J. Physiol. Lung Cell. Mol. Physiol.,2004
5. Analysis of the dissociated steroid RU24858 does not exclude a role for inducible genes in the anti-inflammatory actions of glucocorticoids;Chivers;Mol. Pharmacol.,2006
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