Increased oral sodium chloride intake in humans amplifies selectively postprandial GLP‐1 but not GIP, CCK, and gastrin in plasma

Author:

Asmar Ali12ORCID,Cramon Per K.2,Asmar Meena23,Simonsen Lene2,Sorensen Charlotte M.4ORCID,Madsbad Sten5,Moro Cedric6ORCID,Hartmann Bolette47,Rehfeld Jens F.8,Holst Jens J.47,Hovind Peter12,Jensen Boye L.9,Bülow Jens24

Affiliation:

1. Department of Clinical Physiology, Nuclear Medicine and PET, Rigshospitalet University of Copenhagen Copenhagen Denmark

2. Department of Clinical Physiology and Nuclear Medicine Bispebjerg and Frederiksberg HospitalUniversity Hospital of Copenhagen Copenhagen Denmark

3. Department of Endocrinology Odense University Hospital Odense Denmark

4. Department of Biomedical Sciences University of Copenhagen Copenhagen Denmark

5. Department of Endocrinology Hvidovre HospitalUniversity Hospital of Copenhagen Copenhagen Denmark

6. Institut National de la Santé et de la Recherche Médicale (Inserm) UMR 1048Institute of Metabolic and Cardiovascular DiseasesPaul Sabatier University Toulouse France

7. Novo Nordisk Foundation Center for Basic Metabolic Research University of Copenhagen Copenhagen Denmark

8. Department of Clinical Biochemistry, Rigshospitalet University of Copenhagen Copenhagen Denmark

9. Department of Cardiovascular and Renal Research University of Southern Denmark Odense Denmark

Publisher

Wiley

Subject

Physiology (medical),Physiology

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