PGC‐1α overexpression increases transcription factor EB nuclear localization and lysosome abundance in dystrophin‐deficient skeletal muscle
Author:
Affiliation:
1. Department of Animal Science Iowa State University Ames IA USA
2. Department of Biological Sciences Purdue University West Lafayette IN USA
3. Department of Kinesiology and Applied Physiology University of Delaware Newark DE USA
Funder
American Physiological Society
Parent Project Muscular Dystrophy
Publisher
Wiley
Subject
Physiology (medical),Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.14814/phy2.14383
Reference34 articles.
1. Calcium Influx through Calcium Leak Channels Is Responsible for the Elevated Levels of Calcium-dependent Proteolysis in Dystrophic Myotubes
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4. PGC-1α and PGC-1β Increase Protein Synthesis via ERRα in C2C12 Myotubes
5. Post-natal induction of PGC-1α protects against severe muscle dystrophy independently of utrophin
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