NLRP 3 inflammasome activation in platelets in response to sepsis-Reference-Cited by-同舟云学术

NLRP 3 inflammasome activation in platelets in response to sepsis

Published:2019-05 Issue:9 Volume:7 Page:e14073
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Cornelius Denise C.¹²³^ORCID,Baik Cedar H.¹,Travis Olivia K.²,White Dakota L.¹,Young Cassandra M.²,Austin Pierce W.¹,Shields Corbin A.²,Poudel Bibek²,Williams Jan M.²³

Affiliation:

1. Department of Emergency Medicine University of Mississippi Medical Center Jackson Mississippi

2. Department of Pharmacology University of Mississippi Medical Center Jackson Mississippi

3. Cardiovascular Renal‐Research Center University of Mississippi Medical Center Jackson Mississippi

Funder

National Institute of General Medical Sciences

National Heart, Lung, and Blood Institute

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.14814/phy2.14073

Reference37 articles.

1. NLRP3 inflammasome: From a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases

2. Human platelets produce and constitute the main source of IL‐18 in the circulation (44.25);Allam O.;J. Immunol.,2012

3. Sepsis-Associated Acute Kidney Injury

4. Escaping the Nuclear Confines: Signal-Dependent Pre-mRNA Splicing in Anucleate Platelets

5. Interleukin-1β

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