Alteration of renal Na,K-ATPase in rats following the mediastinal γ-irradiation

Author:

Kaločayová Barbora1,Kovačičová Ivona1,Radošinská Jana12,Tóthová Ľubomíra3,Jagmaševič-Mézešová Lucia1,Fülöp Marko4,Slezák Ján1,Babál Pavel5,Janega Pavol5,Vrbjar Norbert1

Affiliation:

1. Centre of Experimental Medicine; Institute for Heart Research; Slovak Academy of Sciences; Bratislava Slovak Republic

2. Institute of Physiology; Faculty of Medicine; Comenius University in Bratislava; Bratislava Slovak Republic

3. Institute of Molecular Biomedicine; Faculty of Medicine; Comenius University; Bratislava Slovak Republic

4. Slovak Medical University; Bratislava Slovak Republic

5. Institute of Pathology; Faculty of Medicine; Comenius University in Bratislava; Bratislava Slovak Republic

Funder

Slovak Grant Agencies

Publisher

Wiley

Subject

Physiology (medical),Physiology

Reference39 articles.

1. Advanced glycation end products induce apoptosis in fibroblasts through activation of ROS, MAP kinases, and the FOXO1 transcription factor;Alikhani;Am. J. Physiol. Cell Physiol.,2007

2. Isolation and purification of the extracellular and intracellular portions of the beta subunit of (Na+, K+)-ATPase;Ataei;Prep. Biochem.,1992

3. Role of free radicals on mechanism of radiation nephropathy;Balabanli;Mol. Cell. Biochem.,2006

4. Preoperative radiation for retroperitoneal sarcoma is not associated with increased early postoperative morbidity;Bartlett;J. Surg. Oncol.,2014

5. The ferric reducing ability of plasma (FRAP) as a measure of “antioxidant power”: the FRAP assay;Benzie;Anal. Biochem.,1996

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