Paradoxical effect of IKKβ inhibition on the expression of E3 ubiquitin ligases and unloading-induced skeletal muscle atrophy
Author:
Affiliation:
1. Institute of Biomedical Problems, RAS; Moscow Russia
2. Department of Anatomy and Cell Biology; Indiana University School of Medicine-Northwest; Gary Indiana
3. Faculty of Basic Medicine; Lomonosov Moscow State University; Moscow Russia
Funder
Russian Foundation of Basic Research
School of Medicine, Indiana University
Publisher
Wiley
Subject
Physiology (medical),Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.13291/fullpdf
Reference41 articles.
1. Muscle-specific and age-related changes in protein synthesis and protein degradation in response to hindlimb unloading in rats;Baehr;J. Appl. Physiol,2017
2. The effect of hindlimb immobilization on acid phosphatase, metalloproteinases and nuclear factor-kappaB in muscles of young and old rats;Bar-Shai;Mech. Ageing Dev.,2005
3. Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1;Bodine;Am. J. Physiol. Endocrinol. Metab.,2014
4. IKKbeta/NF-kappaB activation causes severe muscle wasting in mice;Cai;Cell,2004
5. PGC1-alpha over-expression prevents metabolic alterations and soleus muscle atrophy in hindlimb unloaded mice;Cannavino;J. Physiol.,2014
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