mTORc1 activity is necessary and sufficient for phosphorylation of eNOSS1177-Reference-Cited by-同舟云学术

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mTORc1 activity is necessary and sufficient for phosphorylation of eNOSS1177

Published:2018-06 Issue:12 Volume:6 Page:e13733
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Decker Brandee¹,Pumiglia Kevin¹^ORCID

Affiliation:

1. Department of Regenerative and Cancer Cell Biology; Albany Medical College; Albany New York

Funder

DOD Neurofibromatosis Program

David E. Bryant Trust

Leopold Fund for Vascular Anomalies

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.13733/fullpdf

Reference47 articles.

1. Activation of endothelial ras signaling bypasses senescence and causes abnormal vascular morphogenesis;Bajaj;Cancer Res.,2010

2. PI3K/mTOR inhibition promotes the regression of experimental vascular malformations driven by PIK3CA-activating mutations;Blasio;Cell Death Dis.,2018

3. Endothelial nitric-oxide synthase (type III) is activated and becomes calcium independent upon phosphorylation by cyclic nucleotide-dependent protein kinases;Butt;J. Biol. Chem.,2000

4. Endothelial dysfunction in cardiovascular diseases: the role of oxidant stress;Cai;Circ. Res.,2000

5. CaM Kinase II-dependent pathophysiological signalling in endothelial cells;Cai;Cardiovasc. Res.,2008

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