LPS ‐stimulated NF ‐ κ B p65 dynamic response marks the initiation of TNF expression and transition to IL ‐10 expression in RAW 264.7 macrophages-Reference-Cited by-同舟云学术

LPS ‐stimulated NF ‐ κ B p65 dynamic response marks the initiation of TNF expression and transition to IL ‐10 expression in RAW 264.7 macrophages

Published:2018-11 Issue:21 Volume:6 Page:
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Hobbs Stuart¹^ORCID,Reynoso Marinaliz¹,Geddis Alyssa V.¹,Mitrophanov Alexander Y.²,Matheny Ronald W.¹

Affiliation:

1. Military Performance Division U.S. Army Research Institute of Environmental Medicine Natick Massachusetts

2. DoD Biotechnology High Performance Computing Software Applications Institute Telemedicine and Advanced Technology Research Center U.S. Army Medical Research and Materiel Command Ft. Detrick Maryland

Funder

Medical Research and Materiel Command

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.14814/phy2.13914

Reference53 articles.

1. Signal transduction controls heterogeneous NF-κB dynamics and target gene expression through cytokine-specific refractory states

2. Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis

3. Tumor Necrosis Factor Alpha Transcription in Macrophages Is Attenuated by an Autocrine Factor That Preferentially Induces NF-κB p50

4. Interleukin-10 is a central regulator of the response to LPS in murine models of endotoxic shock and the Shwartzman reaction but not endotoxin tolerance.

5. Endotoxin tolerance: new mechanisms, molecules and clinical significance

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