Dysregulated insulin secretion is associated with pancreatic β‐cell hyperplasia and direct acinar‐β‐cell trans‐differentiation in partially <scp>eNOS</scp> ‐deficient mice-Reference-Cited by-同舟云学术

Dysregulated insulin secretion is associated with pancreatic β‐cell hyperplasia and direct acinar‐β‐cell trans‐differentiation in partially eNOS ‐deficient mice

Published:2022-08 Issue:16 Volume:10 Page:
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiological Reports

Author:

Novelli Michela¹,Masini Matilde¹,Vecoli Cecilia²,Moscato Stefania³⁴,Funel Niccola¹,Pippa Anna²,Mattii Letizia³⁴,Ippolito Chiara³,Campani Daniela⁵,Neglia Danilo⁶,Masiello Pellegrino¹^ORCID

Affiliation:

1. Department of Translational Research and New Technologies in Medicine and Surgery University of Pisa Pisa Italy

2. Institute of Clinical Physiology National Research Council (CNR) Pisa Italy

3. Department of Clinical and Experimental Medicine University of Pisa Pisa Italy

4. Interdepartmental Research Centre "Nutraceuticals and Food for Health" University of Pisa Pisa Italy

5. Department of Surgical, Medical and Molecular Pathology, and Critical Care Medicine University of Pisa Pisa Italy

6. Cardiovascular Department Fondazione Toscana Gabriele Monasterio per la Ricerca Medica e di Sanità Pubblica Pisa Italy

Funder

Università di Pisa

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.14814/phy2.15425

Reference71 articles.

1. Pancreatic β Cell Regeneration as a Possible Therapy for Diabetes

2. Autophagy negatively regulates tumor cell proliferation through phosphorylation dependent degradation of the Notch1 intracellular domain

3. Natural history of β-cell adaptation and failure in type 2 diabetes

4. Notch Signaling: Cell Fate Control and Signal Integration in Development

5. Notch Signaling as Gatekeeper of Rat Acinar-to-β-Cell Conversion in Vitro