A Potential New Mouse Model of Axial Spondyloarthritis Involving the Complement System
Author:
Affiliation:
1. Division of Rheumatology, School of Medicine, University of Colorado, Anschutz Medical Campus, Aurora, CO 80045, USA.
2. Department of Pathology, School of Medicine, University of Colorado, Anschutz Medical Campus, Aurora, CO 80045, USA.
Publisher
The Korean Association of Immunobiologists
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Link
https://immunenetwork.org/pdf/10.4110/in.2021.21.e45
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1. Trends in diagnostic prevalence and treatment patterns of male and female ankylosing spondylitis patients in the United States, 2006–2016
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3. Additional human β2-microglobulin curbs HLA–B27 misfolding and promotes arthritis and spondylitis without colitis in male HLA–B27–transgenic rats
4. Are spondylarthritides related but distinct conditions or a single disease with a heterogeneous phenotype?
5. IL-23 induces spondyloarthropathy by acting on ROR-γt+ CD3+CD4−CD8− entheseal resident T cells
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1. The complement factor H-related protein-5 (CFHR5) exacerbates pathological bone formation in ankylosing spondylitis;Journal of Molecular Medicine;2024-02-29
2. An update of murine models and their methodologies in immune-mediated joint damage and pain research;International Immunopharmacology;2024-02
3. One Autoimmune Disease Might Pave Way for Another;Lippincott's Bone and Joint Newsletter;2022-05
4. The Complement System in Spondyloarthritis: What Do We Know?;Rheumatology;2022
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