Origin and Development of the Precursor Lesions in Experimental Pancreatic Cancer in Rats-Reference-Cited by-同舟云学术

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Origin and Development of the Precursor Lesions in Experimental Pancreatic Cancer in Rats

Published:2003-06 Issue:6 Volume:83 Page:853-859
ISSN:0023-6837
Container-title:Laboratory Investigation
language:
Short-container-title:Lab Invest

Author:

Bockman Dale E,Guo Junchao,Büchler Peter,Müller Michael W,Bergmann Frank,Friess Helmut

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Reference28 articles.

1. Cytological changes in the pancreas of transgenic mice overexpressing transforming growth factor α

2. Fine Structure of Pancreatic Adenocarcinoma Induced In Rats by 7,12-Dimethylbenz[ a ]anthracene 2

3. Origin of tubular complexes in human chronic pancreatitis

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1. The E3 ubiquitin ligase TRIP12 is required for pancreatic acinar cell plasticity and pancreatic carcinogenesis;The Journal of Pathology;2024-06-25

2. The Multifaceted Role of miR-21 in Pancreatic Cancers;Cells;2024-05-30

3. Expression of BAX and eNOS in Rabbit Pancreatic Tissues Injured by Hydrocortisone;Al-Rafidain Journal of Medical Sciences ( ISSN 2789-3219 );2024-02-22

4. Advances in tuft cells, a chemosensory cell in sequential diseases of the pancreas;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2023-07

5. The Loss of the E3 ubiquitin ligase TRIP12 inhibits Pancreatic Acinar Cell Plasticity and Tumor Cell Metastatic Capacity;2023-03-10

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