Enterococcus faecalis Exacerbates Burn Injury-Induced Host Responses in Rats-Reference-Cited by-同舟云学术

Enterococcus faecalis Exacerbates Burn Injury-Induced Host Responses in Rats

Published:2002-12 Issue:6 Volume:18 Page:523-528
ISSN:1073-2322
Container-title:Shock
language:en
Short-container-title:Shock

Author:

Goto Masakatsu,Samonte Victoria,Khan Mehdi,Haque Farah,Goyal Amit,Al-Ghoul Walid,Raziuddin Syed,Fazal Nadeem,Ravindranath Thyyar,Reed R. Lawrence,Gamelli Richard L.,Sayeed Mohammed M.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference29 articles.

1. Selective decontamination of the digestive tract in severely burned pediatric patients

2. Burn Wound Infections: Current Status

3. ROLE OF NEUTROPHILS IN BURN-INDUCED MICROVASCULAR INJURY IN THE INTESTINE

4. Neutrophil depletion prevents intestinal mucosal permeability alterations in burn-injured rats

5. Neutrophil depletion in rats reduces burn-injury induced intestinal bacterial translocation

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1. Bacterial nanocellulose loaded with bromelain and nisin as a promising bioactive material for wound debridement;International Journal of Biological Macromolecules;2024-05

2. Differential effector responses by circulating/blood and tissue/peritoneal neutrophils following burn combined with Enterococcus faecalis infection;FEMS Immunology & Medical Microbiology;2011-11-21

3. Burn Injury Exacerbates Hemodynamic and Metabolic Responses in Rats with Polymicrobial Sepsis;Journal of Burn Care & Research;2006-01

4. INFLAMMATORY/CARDIOVASCULAR-METABOLIC RESPONSES IN A RAT MODEL OF BURN INJURY WITH SUPERIMPOSED INFECTION;Shock;2005-12

5. Exacerbation of intestinal permeability in rats after a two-hit injury: Burn and Enterococcus faecalis infection*;Critical Care Medicine;2004-11