Concomitant BCR-ABL1 Translocation and JAK2V617F Mutation in Three Patients with Myeloproliferative Neoplasms-Reference-Cited by-同舟云学术

Concomitant BCR-ABL1 Translocation and JAK2V617F Mutation in Three Patients with Myeloproliferative Neoplasms

Published:2012-09 Issue:3 Volume:21 Page:176-183
ISSN:1052-9551
Container-title:Diagnostic Molecular Pathology
language:en
Short-container-title:Diagnostic Molecular Pathology

Author:

Hummel Jennifer M.,Kletecka M. Carmen Frias,Sanks Jennifer K.,Chiselite Mihaela D.,Roulston Diane,Smith Lauren B.,Czuchlewski David R.,Elenitoba-Johnson Kojo S. J.,Lim Megan S.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Reference27 articles.

1. A Gain-of-Function Mutation ofJAK2in Myeloproliferative Disorders

2. The hematopoietic stem cell compartment of JAK2V617F-positive myeloproliferative disorders is a reflection of disease heterogeneity

3. Chronic myeloproliferative diseases with concurrent BCR–ABL junction and JAK2V617F mutation

4. BCR-ABL1-positive CML and BCR-ABL1-negative chronic myeloproliferative disorders: some common and contrasting features

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5. Sequential Development of JAK2V617F Mutation and BCR-ABL1 Fusion in Individual Patients With Myeloproliferative Neoplasms: A Linear Clonal Evolution or Parallel Clonal Competition?;Archives of Pathology & Laboratory Medicine;2021-09-10