Regulation of Bcl-2 Expression by Dihydrotestosterone in Hormone Sensitive LNCaP-FGC Prostate Cancer Cells
Author:
Affiliation:
1. From the Departments of Molecular Pathology and Genitourinary Medical Oncology, University of Texas M. D. Anderson Cancer Center and Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Urology
Reference19 articles.
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2. A critical analysis of tumor morphology and hormone treatments in the untreated and estrogen-treated responsive and refractory human prostatic carcinoma;Sinha;Cancer,1977
3. Deletion of cells by apoptosis during castration-induced involution of the rat prostate;Kerr;Virchows Arch B Cell Pathol,1973
4. Expression of the protooncogene bcl-2 in the prostate and its association with emergence of androgen-independent prostate cancer;McDonnell;Cancer Res,1992
5. Overexpression of bcl-2 protects prostate cancer cells from apoptosis in vitro and confers resistance to androgen depletion in vivo;Raffo;Cancer Res,1995
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