GTS-21 Reduces Inflammation in Acute Lung Injury by Regulating M1 Polarization and Function of Alveolar Macrophages-Reference-Cited by-同舟云学术

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GTS-21 Reduces Inflammation in Acute Lung Injury by Regulating M1 Polarization and Function of Alveolar Macrophages

Published:2019-03 Issue:3 Volume:51 Page:389-400
ISSN:1073-2322
Container-title:Shock
language:en
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Author:

Wang Jing,Li Ruiting,Peng Zhiyong,Zhou Wenhai,Hu Bo,Rao Xin,Yang Xiao,Li Jianguo

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference34 articles.

1. The immunopathogenesis of sepsis;Cohen;Nature,2002

2. Inflammatory processes during acute respiratory distress syndrome: a complex system;Reiss;Curr Opin Crit Care,2018

3. Approaches and techniques to avoid development or progression of acute respiratory distress syndrome;Beloncle;Curr Opin Crit Care,2018

4. The cholinergic anti-inflammatory pathway: a critical review;Martelli;Auton Neurosci,2014

5. The alpha7 nicotinic acetylcholine receptor on fibroblast-like synoviocytes and in synovial tissue from rheumatoid arthritis patients: a possible role for a key neurotransmitter in synovial inflammation;van Maanen;Arthritis Rheum,2009

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1. Molecular mechanisms of Sepsis attacking the immune system and solid organs;Frontiers in Medicine;2024-08-29

2. Retrospective evaluation of COVID-19 incidence during smoking cessation treatment with varenicline;Celal Bayar Üniversitesi Sağlık Bilimleri Enstitüsü Dergisi;2024-03-27

3. GTS-21 attenuates ACE/ACE2 ratio and glycocalyx shedding in lipopolysaccharide-induced acute lung injury by targeting macrophage polarization derived ADAM-17;International Immunopharmacology;2024-03

4. Paralemmin-3 augments lipopolysaccharide-induced acute lung injury with M1 macrophage polarization via the notch signaling pathway;Respiratory Physiology & Neurobiology;2024-02

5. Cholinergic signaling via the α7 nicotinic acetylcholine receptor regulates the migration of monocyte-derived macrophages during acute inflammation;Journal of Neuroinflammation;2024-01-04

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