Helicobacter pylori infection: pathogenesis
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Gastroenterology
Reference49 articles.
1. Functional Analysis of the Helicobacter pylori cag Pathogenicity Island Reveals Both VirD4-CagA-Dependent and VirD4-CagA-Independent Mechanisms
2. Systematic mutagenesis of the Helicobacter pylori cag pathogenicity island: essential genes for CagA translocation in host cells and induction of interleukin-8
3. Phosphorylation of tyrosine 972 of the Helicobacter pylori CagA protein is essential for induction of a scattering phenotype in gastric epithelial cells
4. SHP-2 Tyrosine Phosphatase as an Intracellular Target of Helicobacter pylori CagA Protein
5. ZBP-89, Sp1, and Nuclear Factor-κB Regulate Epithelial Neutrophil-activating Peptide-78 Gene Expression in Caco-2 Human Colonic Epithelial Cells
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1. Association between Helicobacter pylori and liver-to-spleen ratio;European Journal of Gastroenterology & Hepatology;2013-01
2. Suppression of IL-8 production in gastric epithelial cells by MUC1 mucin and peroxisome proliferator-associated receptor-γ;American Journal of Physiology-Gastrointestinal and Liver Physiology;2012-09-15
3. Helicobacter pylori and Gastric Cancer: Factors That Modulate Disease Risk;Clinical Microbiology Reviews;2010-10
4. Role of Innate Immunity inHelicobacter pylori-Induced Gastric Malignancy;Physiological Reviews;2010-07
5. Muc1 Cell Surface Mucin Attenuates Epithelial Inflammation in Response to a Common Mucosal Pathogen;Journal of Biological Chemistry;2010-07
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