Central amygdala inflammation drives pain hypersensitivity and attenuates morphine analgesia in experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

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Central amygdala inflammation drives pain hypersensitivity and attenuates morphine analgesia in experimental autoimmune encephalomyelitis

Published:2021-04-09 Issue:1 Volume:163 Page:e49-e61
ISSN:0304-3959
Container-title:Pain
language:en
Short-container-title:PAIN

Author:

Dworsky-Fried Zoë,Faig Christian A.,Vogel Holly A.,Kerr Bradley J.,Taylor Anna M.W.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Neurology (clinical),Neurology

Reference90 articles.

1. Reduced microglial activity and enhanced glutamate transmission in the basolateral amygdala in early CNS autoimmunity;Acharjee;J Neurosci,2018

2. Cell-type specificity of neuronal excitability and morphology in the central amygdala;Adke;eNeuro,2021

3. Lack of analgesic effect of opioids on neuropathic and idiopathic forms of pain;Arnér;PAIN,1988

4. Sex-related differences in the effects of morphine and stress on visceral pain;Baamonde;Neuropharmacology,1989

5. Astrogliosis in EAE spinal cord: derivation from radial glia, and relationships to oligodendroglia;Bannerman;Glia,2007

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4. Endogenous opiates and behavior: 2022;Peptides;2023-11

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