An NPY Y1 receptor antagonist unmasks latent sensitization and reveals the contribution of protein kinase A and Epac to chronic inflammatory pain-Reference-Cited by-同舟云学术

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An NPY Y1 receptor antagonist unmasks latent sensitization and reveals the contribution of protein kinase A and Epac to chronic inflammatory pain

Published:2019-03-08 Issue:8 Volume:160 Page:1754-1765
ISSN:0304-3959
Container-title:Pain
language:en
Short-container-title:PAIN

Author:

Fu Weisi,Nelson Tyler S.,Santos Diogo F.,Doolen Suzanne,Gutierrez Javier J.P.,Ye Na,Zhou Jia,K. Taylor Bradley

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Neurology (clinical),Neurology

Reference79 articles.

1. Role of protein kinase A in the maintenance of inflammatory pain;Aley;J Neurosci,1999

2. A novel EPAC-specific inhibitor suppresses pancreatic cancer cell migration and invasion;Almahariq;Mol Pharmacol,2013

3. Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and A-dependent mechanisms in rats and mice;Amadesi;J Physiol,2006

4. Repeated mu-opioid exposure induces a novel form of the hyperalgesic priming model for transition to chronic pain;Araldi;J Neurosci,2015

5. cAMP-dependent protein kinase regulates desensitization of the capsaicin receptor (VR1) by direct phosphorylation;Bhave;Neuron,2002

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