Trauma-Hemorrhagic Shock Mesenteric Lymph Induces Endothelial Apoptosis That Involves Both Caspase-Dependent and Caspase-Independent Mechanisms-Reference-Cited by-同舟云学术

Trauma-Hemorrhagic Shock Mesenteric Lymph Induces Endothelial Apoptosis That Involves Both Caspase-Dependent and Caspase-Independent Mechanisms

Published:2004-07 Issue:1 Volume:240 Page:123-131
ISSN:0003-4932
Container-title:Annals of Surgery
language:en
Short-container-title:Annals of Surgery

Author:

Davidson Marson T.,Deitch Edwin A.,Lu Qi,Hask?? Gy??rgy,Abungu Billy,N??meth Zolt??n H.,Zaets Sergey B.,Gaspers Lawrence D.,Thomas Andrew P.,Xu Da-Zhong

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Surgery

Reference32 articles.

1. Multiple Organ Failure Pathophysiology and Potential Future Therapy

2. Gut-Derived Mesenteric Lymph but not Portal Blood Increases Endothelial Cell Permeability and Promotes Lung Injury After Hemorrhagic Shock

3. A time course study of the protective effect of mesenteric lymph duct ligation on hemorrhagic shock-induced pulmonary injury and the toxic effects of lymph from shocked rats on endothelial cell monolayer permeability

4. MESENTERIC LYMPH DUCT LIGATION PROVIDES LONG TERM PROTECTION AGAINST HEMORRHAGIC SHOCK-INDUCED LUNG INJURY

5. POST-HEMORRHAGIC SHOCK MESENTERIC LYMPH IS CYTOTOXIC TO ENDOTHELIAL CELLS AND ACTIVATES NEUTROPHILS

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