Protein Kinase C Isoforms Responsible for the Regulation of Vascular Calcium Sensitivity and Their Relationship to Integrin-Linked Kinase Pathway After Hemorrhagic Shock-Reference-Cited by-同舟云学术

Protein Kinase C Isoforms Responsible for the Regulation of Vascular Calcium Sensitivity and Their Relationship to Integrin-Linked Kinase Pathway After Hemorrhagic Shock

Published:2010-11 Issue:5 Volume:69 Page:1274-1281
ISSN:0022-5282
Container-title:Journal of Trauma: Injury, Infection & Critical Care
language:en
Short-container-title:

Author:

Xu Jing,Li Tao,Yang Guang-Ming,Liu Liang-Ming

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Surgery

Reference26 articles.

1. The effects of Escherichia coli sepsis and short-term ischemia on coronary vascular reactivity and myocardial function.;Hasan;Shock,1997

2. Hemorrhagic shock–induced vascular hyporeactivity in the rat: relationship to gene expression of nitric oxide, endothline-1, and select cytokines in corresponding organs.;Liu;J Surg Res,2005

3. Opioid receptor antagonists modulate Ca2+-activated K+ channels in mesenteric arterial smooth muscle cells of rats in hemorrhagic shock.;Kai;Shock,2003

4. The role of calcium desensitization in vascular hyporeactivity and its regulation following hemorrhagic shock in the rat.;Xu;Shock,2005

5. Protein kinase C and the regulation of the actin cytoskeleton.;Christer;Cell Signal,2006

Cited by 22 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Methylation-reprogrammed CHRM3 results in vascular dysfunction in the human umbilical vein following IVF-ET;Biology of Reproduction;2021-12-22

2. Hemorrhagic Shock and the Microvasculature;Comprehensive Physiology;2017-12-12

3. TCR signalling network organization at the immunological synapses of murine regulatory T cells;European Journal of Immunology;2017-09-12

4. Calcium Desensitization Mechanism and Treatment for Vascular Hyporesponsiveness After Shock;Advanced Trauma and Surgery;2016-11-16

5. Diversity of Vascular Reactivity and the Treatment Response in Diabetic, Hypertensive, Hyperlipidemic, and Healthy Rats Subjected to Hemorrhagic Shock;Shock;2016-02