Increased extracellular heat shock protein 90α in severe sepsis and SIRS associated with multiple organ failure and related to acute inflammatory-metabolic stress response in children-Reference-Cited by-同舟云学术

Increased extracellular heat shock protein 90α in severe sepsis and SIRS associated with multiple organ failure and related to acute inflammatory-metabolic stress response in children

Published:2016-08 Issue:35 Volume:95 Page:e4651
ISSN:0025-7974
Container-title:Medicine
language:en
Short-container-title:

Author:

Fitrolaki Michaela-Diana,Dimitriou Helen,Venihaki Maria,Katrinaki Marianna,Ilia Stavroula,Briassoulis George

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

General Medicine

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4. Cerebrolysin treatment attenuates heat shock protein overexpression in the brain following heat stress: an experimental study using immunohistochemistry at light and electron microscopy in the rat;Sharma;Ann NY Acad Sci,2010

5. Heat shock protein 72 expressing stress in sepsis: unbridgeable gap between animal and human studies—a hypothetical “comparative” study;Briassoulis;BioMed Res Int,2014

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