Differences in Renal Tubular Na-K-Adenosine Triphosphatase in Spontaneously Hypertensive and Normotensive Rats
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Pharmacology
Cited by 9 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling;International Journal of Nephrology;2012
2. Increases in transepithelial vectorial Na+ transport facilitates Na+-dependent l-DOPA transport in renal OK cells;Life Sciences;2006-07
3. Upregulation of apical NHE3 in renal OK cells overexpressing the rodent α1-subunit of the Na+pump;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2006-04
4. Chronic renal injury-induced hypertension alters renal NHE3 distribution and abundance;American Journal of Physiology-Renal Physiology;2003-05-01
5. Proximal tubule Na transporter responses are the same during acute and chronic hypertension;American Journal of Physiology-Renal Physiology;2000-08-01
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