Ethanol Selectively Counteracts Hypotension Evoked by Central I1-Imidazoline but Not α2-Adrenergic Receptor Activation in Spontaneously Hypertensive Rats
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Pharmacology
Reference43 articles.
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1. Differential Central NOS-NO Signaling Underlies Clonidine Exacerbation of Ethanol-Evoked Behavioral Impairment;Alcoholism: Clinical and Experimental Research;2010-03
2. Direct evidence for imidazoline (I-1) receptor modulation of ethanol action on norepinephrine-containing neurons in the rostral ventrolateral medulla in conscious spontaneously hypertensive rats;ALCOHOL CLIN EXP RES;2007
3. Direct Evidence for Imidazoline (I1) Receptor Modulation of Ethanol Action on Norepinephrine-Containing Neurons in the Rostral Ventrolateral Medulla in Conscious Spontaneously Hypertensive Rats;Alcoholism: Clinical and Experimental Research;2007-02-27
4. Mitogen-Activated Protein Kinase Phosphorylation in the Rostral Ventrolateral Medulla Plays a Key Role in Imidazoline (I1)-Receptor-Mediated Hypotension;Journal of Pharmacology and Experimental Therapeutics;2005-05-18
5. Chronic ethanol administration attenuates imidazoline I1 receptor- or α2-adrenoceptor-mediated reductions in blood pressure and hemodynamic variability in hypertensive rats;European Journal of Pharmacology;2004-02
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