Targeted Deletion of Angiotensin II Type 1A Receptor Does not Protect Mice from Progressive Nephropathy of Overload Proteinuria
Author:
Publisher
American Society of Nephrology (ASN)
Subject
Nephrology,General Medicine
Cited by 28 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Intrarenal Renin–Angiotensin System Involvement in the Pathogenesis of Chronic Progressive Nephropathy—Bridging the Informational Gap Between Disciplines;Toxicologic Pathology;2019-07-07
2. Palmitate deranges erythropoietin production via transcription factor ATF4 activation of unfolded protein response;Kidney International;2018-09
3. Proteinuria and progression of pediatric chronic kidney disease: lessons from recent clinical studies;Pediatric Nephrology;2016-06-27
4. Hypercholesterolemia blunts the oxidative stress elicited by hypertension in venules through angiotensin II type-2 receptors;Microvascular Research;2016-05
5. Therapeutic potential of endothelin receptor antagonism in kidney disease;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2016-03-01
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