Re: comment from Ladak et al. to JH-D-23-00692, entitled ‘Analyzing eGFR-Dip after medical target therapy in primary aldosteronism’
Author:
Affiliation:
1. Department of Urology, National Taiwan University Hospital
2. Division of Nephrology, Department of Internal Medicine, National Taiwan University Hospital, Taiwan
Publisher
Ovid Technologies (Wolters Kluwer Health)
Reference7 articles.
1. Estimated glomerular filtration rate-dip after medical target therapy associated with increased mortality and cardiovascular events in patients with primary aldosteronism;Sheu;J Hypertens,2023
2. Divergent characteristics of T-cell receptor repertoire between essential hypertension and aldosterone-producing adenoma;Chang;Front Immunol,2022
3. Markers of kidney tubular function deteriorate while those of kidney tubule health improve in primary aldosteronism after targeted treatments;Wu;J Am Heart Assoc,2023
4. Primary aldosteronism: changes in cystatin C-based kidney filtration, proteinuria, and renal duplex indices with treatment;Wu;J Hypertens,2011
5. Kidney function predicts new-onset cardiorenal events and mortality in primary aldosteronism: approach of the 2021 race-free eGFR equation;Lai;Hypertens Res,2023
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