Disruption of the with no lysine kinase–STE20-proline alanine-rich kinase pathway reduces the hypertension induced by angiotensin II-Reference-Cited by-同舟云学术

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Disruption of the with no lysine kinase–STE20-proline alanine-rich kinase pathway reduces the hypertension induced by angiotensin II

Published:2018-02 Issue:2 Volume:36 Page:361-367
ISSN:0263-6352
Container-title:Journal of Hypertension
language:en
Short-container-title:

Author:

Cervantes-Perez Luz G.,Castaneda-Bueno Maria,Jimenez Jose V.,Vazquez Norma,Rojas-Vega Lorena,Alessi Dario R.,Bobadilla Norma A.,Gamba Gerardo

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine

Reference32 articles.

1. Angiotensin II causes hypertension and cardiac hypertrophy through its receptors in the kidney;Crowley;Proc Natl Acad Sci U S A,2006

2. Role of AT(1) receptor-mediated salt retention in angiotensin II-dependent hypertension;Crowley;Am J Physiol Renal Physiol,2011

3. The absence of intrarenal ACE protects against hypertension;Gonzalez-Villalobos;J Clin Invest,2013

4. Enhanced distal nephron sodium reabsorption in chronic angiotensin II-infused mice;Zhao;Hypertension,2009

5. Acute angiotensin II infusions elicit pressure natriuresis in mice and reduce distal fractional sodium reabsorption;Zhao;Hypertension,2008

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Inactivation of SPAK kinase reduces body weight gain in mice fed a high-fat diet by improving energy expenditure and insulin sensitivity;American Journal of Physiology-Endocrinology and Metabolism;2018-01-01

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