Trauma-Hemorrhagic Shock Induces a CD36-Dependent RBC Endothelial-Adhesive Phenotype-Reference-Cited by-同舟云学术

Trauma-Hemorrhagic Shock Induces a CD36-Dependent RBC Endothelial-Adhesive Phenotype

Published:2014-03 Issue:3 Volume:42 Page:e200-e210
ISSN:0090-3493
Container-title:Critical Care Medicine
language:en
Short-container-title:Critical Care Medicine

Author:

Deitch Edwin A.,Condon Michael,Feketeova Eleonora,Machiedo George W.,Mason Leonard,Vinluan Ghia M.,Alli Vamsi A.,Neal Matthew D.,Tomaio Jacquelyn N.,Fishman Jordan E.,Durán Walter N.,Spolarics Zoltan

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine

Reference40 articles.

1. Postinjury multiple organ failure

2. Molecular basis of sickle cell-endothelial cell interactions

3. Advanced glycation end products (AGEs) on the surface of diabetic erythrocytes bind to the vessel wall via a specific receptor inducing oxidant stress in the vasculature: a link between surface-associated AGEs and diabetic complications.

4. Mesenteric lymph duct ligation prevents shock-induced RBC deformability and shape changes

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