LACK OF COGNATE HELP BY CD4+ T CELLS AND ANERGY OF CD8+ T CELLS ARE THE PRINCIPAL MECHANISMS FOR ANTI-LEUKOCYTE FUNCTION-ASSOCIATED ANTIGEN-1/INTERCELLULAR ADHESION MOLECULE-1-INDUCED CARDIAC ALLOGRAFT TOLERANCE
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference24 articles.
1. Clonal Expansion Versus Functional Clonal Inactivation: A Costimulatory Signalling Pathway Determines the Outcome of T Cell Antigen Receptor Occupancy
2. Failure of clonal deletion in neonatally thymectomized mice: tolerance is preserved through clonal anergy.
3. B7 but not intercellular adhesion molecule-1 costimulation prevents the induction of human alloantigen-specific tolerance.
4. Specific Acceptance of Cardiac Allograft After Treatment with Antibodies to ICAM-1 and LFA-1
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1. Lymph Node-Targeted Synthetically Glycosylated Antigen Leads to Antigen-Specific Immunological Tolerance;Frontiers in Immunology;2021-09-24
2. Anti-LFA-1 induces CD8 T-cell dependent allograft tolerance and augments suppressor phenotype CD8 cells;Cellular Immunology;2018-10
3. Approaches to the Induction of Tolerance;Kidney Transplantation;2008
4. CD4+ CD25+ regulatory cells in acquired MHC tolerance;Immunological Reviews;2001-08
5. Requirement for natural killer T (NKT) cells in the induction of allograft tolerance;Proceedings of the National Academy of Sciences;2001-02-20
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