ADDITIVE EFFICACY OF CTLA4Ig AND OX40Ig SECRETED BY GENETICALLY MODIFIED GRAFTS1
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference27 articles.
1. INHIBITION OF TRANSPLANT REJECTION FOLLOWING TREATMENT WITH ANTI-B7-2 AND ANTI-B7-1 ANTIBODIES
2. T-cell activation by the CD28 ligand B7 is required for cardiac allograft rejection in vivo.
3. CTLA4Ig TREATMENT AMELIORATES THE LETHALITY OF MURINE GRAFT-VERSUS-HOST DISEASE ACROSS MAJOR HISTOCOMPATIBILITY COMPLEX BARRIERS
4. TRANSPLANTATION TOLERANCE INDUCED BY CTLA4-Ig1
5. Long-term acceptance of major histocompatibility complex mismatched cardiac allografts induced by CTLA4Ig plus donor-specific transfusion.
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1. Adenovirus-mediated OX40Ig gene transfer induces long-term survival of orthotopic liver allograft in rats;Transplant Immunology;2018-06
2. Effect of combined OX40Ig and CTLA4Ig gene local transfer on allograft rejection and the underlying mechanisms;Journal of Surgical Research;2012-12
3. Use of Genetically Modified Allograft to Deliver Local Immunomodulatory Molecule with Minimal Systemic Toxicity in a Rat Model of Allogeneic Skin Flap Transplantation;Transplantation Proceedings;2010-11
4. Dendritic cells expressing soluble CTLA4Ig prolong antigen‐specific skin graft survival;Immunology & Cell Biology;2010-04-20
5. Gene Therapy for Type 1 Diabetes;American Journal of Therapeutics;2005-11
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