Shortening of cardiac action potentials in endotoxic shock in guinea pigs is caused by an increase in nitric oxide activity and activation of the adenosine triphosphate-sensitive potassium channel-Reference-Cited by-同舟云学术

Shortening of cardiac action potentials in endotoxic shock in guinea pigs is caused by an increase in nitric oxide activity and activation of the adenosine triphosphate-sensitive potassium channel

Published:2000-06 Issue:6 Volume:28 Page:1713-1720
ISSN:0090-3493
Container-title:Critical Care Medicine
language:en
Short-container-title:Critical Care Medicine

Author:

Chen Chien-Cheng,Lin Yu-Chin,Chen Shih-Ann,Luk Hsiang-Ning,Ding Philip Yu-An,Chang Mau-Song,Chiang Chern-En

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine

Reference67 articles.

1. Nitric oxide: mediator, murderer, and medicine

2. Nitric Oxide and Cardiac Function

3. Nitric Oxide-dependent Parasympathetic Signaling Is Due to Activation of Constitutive Endothelial (Type III) Nitric Oxide Synthase in Cardiac Myocytes

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