Toll-like receptor distribution in colonic epithelium and lamina propria is disrupted in HIV viremic, immune success, and failure-Reference-Cited by-同舟云学术

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Toll-like receptor distribution in colonic epithelium and lamina propria is disrupted in HIV viremic, immune success, and failure

Published:2020-05-01 Issue:6 Volume:34 Page:815-826
ISSN:0269-9370
Container-title:AIDS
language:en
Short-container-title:

Author:

McCausland Marie R.,Cruz-Lebrón Angélica,Pilch-Cooper Heather A.,Howell Scott,Albert Jeffrey M.,Park Young S.,Levine Alan D.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference49 articles.

1. Gut epithelial barrier dysfunction and innate immune activation predict mortality in treated HIV infection;Hunt;J Infect Dis,2014

2. Disruption of intestinal CD4(+) T cell homeostasis is a key marker of systemic CD4(+) T cell activation in HIV-infected individuals;Gordon;J Immunol,2010

3. Severe CD4+ T-cell depletion in gut lymphoid tissue during primary human immunodeficiency virus type 1 infection and substantial delay in restoration following highly active antiretroviral therapy;Guadalupe;J Virol,2003

4. Antiviral therapy during primary simian immunodeficiency virus infection fails to prevent acute loss of CD4+ T cells in gut mucosa but enhances their rapid restoration through central memory T cells;Verhoeven;J Virol,2008

5. Early antiretroviral therapy for simian immunodeficiency virus infection leads to mucosal CD4+ T-cell restoration and enhanced gene expression regulating mucosal repair and regeneration;George;J Virol,2005

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