Neuroinflammatory Gene Regulation, Mitochondrial Function, Oxidative Stress, and Brain Lipid Modifications With Disease Progression in Tau P301S Transgenic Mice as a Model of Frontotemporal Lobar Degeneration-Tau-Reference-Cited by-同舟云学术

Neuroinflammatory Gene Regulation, Mitochondrial Function, Oxidative Stress, and Brain Lipid Modifications With Disease Progression in Tau P301S Transgenic Mice as a Model of Frontotemporal Lobar Degeneration-Tau

Published:2015-10 Issue:10 Volume:74 Page:975-999
ISSN:0022-3069
Container-title:Journal of Neuropathology & Experimental Neurology
language:en
Short-container-title:J Neuropathol Exp Neurol

Author:

López-González Irene,Aso Ester,Carmona Margarita,Armand-Ugon Mercedes,Blanco Rosi,Naudí Alba,Cabré Rosanna,Portero-Otin Manuel,Pamplona Reinald,Ferrer Isidre

Publisher

Oxford University Press (OUP)

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Neurology,General Medicine,Pathology and Forensic Medicine

Link

http://academic.oup.com/jnen/article-pdf/74/10/975/17420029/74-10-975.pdf

Reference45 articles.

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3. Invited review: Frontotemporal dementia caused by microtubule‐associated protein tau gene ( MAPT ) mutations: a chameleon for neuropathology and neuroimaging

4. Glial and Neuronal Tau Pathology in Tauopathies

5. Dujardin S Colin M Buée L . Invited review: Animal models of tauopathies and their implications for research/translation into the clinic. Neuropathol Appl Neurobiol 2015;41:59–80

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