Reduced Alloreactive T-Cell Activation After Alcohol Intake is Due to Impaired Monocyte Accessory Cell Function and Correlates With Elevated IL-10, IL-13, and Decreased IFN?? Levels-Reference-Cited by-同舟云学术

Reduced Alloreactive T-Cell Activation After Alcohol Intake is Due to Impaired Monocyte Accessory Cell Function and Correlates With Elevated IL-10, IL-13, and Decreased IFN?? Levels

Published:2001-12 Issue:12 Volume:25 Page:1766-1772
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcoholism: Clinical and Experimental Research

Author:

Szabo Gyongyi,Mandrekar Pranoti,Dolganiuc Angela,Catalano Donna,Kodys Karen

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1097/00000374-200112000-00012/fullpdf

Reference47 articles.

1. IL-10 prevents the differentiation of monocytes to dendritic cells but promotes their maturation to macrophages

2. Acute alcohol intoxication suppresses natural killer cell activity and promotes tumor metastasis

3. Initial and innate responses to viral infections — pattern setting in immunity or disease

4. Ethanol Impairs Major Histocompatibility Complex (MHC) Class II Molecule-Mediated But not MHC Class I Molecule-Mediated T Cell Response in Alcohol-Consuming Mice

5. Ethanol modulation of tumor necrosis factor and gamma interferon production by murine splenocytes and macrophages

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