Improvement of renal dysfunction in rats with chronic heart failure after myocardial infarction by treatment with the endothelin A receptor antagonist, LU 135252-Reference-Cited by-同舟云学术

Improvement of renal dysfunction in rats with chronic heart failure after myocardial infarction by treatment with the endothelin A receptor antagonist, LU 135252

Published:2000-10 Issue:10 Volume:18 Page:1507-1514
ISSN:0263-6352
Container-title:Journal of Hypertension
language:en
Short-container-title:Journal of Hypertension

Author:

Bauersachs Johann,Braun Claude,Fraccarollo Daniela,Widdera Julian,Ertl Georg,Schilling Lothar,Kirchengast Michael,Rohmeiss Peter

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine

Reference34 articles.

1. Endothelin in human congestive heart failure.

2. Evidence for endothelin-1-mediated vasoconstriction in severe chronic heart failure

3. Role of endothelin in the maintenance of blood pressure in conscious rats with chronic heart failure. Acute effects of the endothelin receptor antagonist Ro 47-0203 (bosentan).

4. Chronic Endothelin Receptor Blockade Attenuates Progressive Ventricular Dilation and Improves Cardiac Function in Rats With Myocardial Infarction

5. Role of Endogenous Endothelin in Chronic Heart Failure

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1. Edema formation in congestive heart failure and the underlying mechanisms;Frontiers in Cardiovascular Medicine;2022-09-27

2. A Clinically Relevant Functional Model of Type-2 Cardio-Renal Syndrome with Paraventricular Changes consequent to Chronic Ischaemic Heart Failure;Scientific Reports;2020-01-27

3. Patients with hypertensive responses to exercise or dobutamine stress testing differ in resting hypertensive phenotype;Journal of the American Society of Hypertension;2018-02

4. Involvement of Cytokines in the Pathogenesis of Salt and Water Imbalance in Congestive Heart Failure;Frontiers in Immunology;2017-06-19

5. Cardiorenal syndrome: acute kidney injury secondary to cardiovascular disease and role of protein-bound uraemic toxins;The Journal of Physiology;2014-06-25