YKL40 potentially via activating the P38 pathway in decreasing atherosclerotic plaque stability-Reference-Cited by-同舟云学术

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YKL40 potentially via activating the P38 pathway in decreasing atherosclerotic plaque stability

Published:2024-03-26 Issue:8 Volume:137 Page:1003-1005
ISSN:0366-6999
Container-title:Chinese Medical Journal
language:en
Short-container-title:

Author:

Wang Chao¹,Liu Yandong¹²,Huan Wei¹,Wu Jianjin¹,Jin Jie¹,Zou Sili¹,Chen Yu³,Qu Lefeng¹

Affiliation:

1. Department of Vascular and Endovascular Surgery, Changzheng Hospital Affiliated to the Naval Medical University, Shanghai 200003, China

2. Department of Emergency, Navy 905th Hospital, Shanghai 200050, China

3. Clinical Research Institute of Integrative Medicine, Yueyang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 200437, China

Publisher

Ovid Technologies (Wolters Kluwer Health)

Reference6 articles.

1. Global and national burden of atherosclerosis from 1990 to 2019: trend analysis based on the Global Burden of Disease Study 2019;Chen;Chin Med J,2023

2. Research progress on the risk factors and outcomes of human carotid atherosclerotic plaques;Xiong;Chin Med J,2017

3. Role of vascular smooth muscle cell plasticity and interactions in vessel wall inflammation;Sorokin;Front Immunol,2020

4. The role of smooth muscle cells in plaque stability: Therapeutic targeting potential;Harman;Br J Pharmacol,2019

5. Chitinase 3 like 1 is a regulator of smooth muscle cell physiology and atherosclerotic lesion stability;Tsantilas;Cardiovasc Res,2021

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