N-acetyl-aspartylglutamate (NAAG) elicits rapid increase in intraneuronal Ca2+ in vitro
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
General Neuroscience
Cited by 25 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Glycine release is regulated by metabotropic glutamate receptors sensitive to mGluR2/3 ligands and activated by N-acetylaspartylglutamate (NAAG);Neuropharmacology;2013-03
2. The effect of N-acetyl-aspartyl-glutamate and N-acetyl-aspartate on white matter oligodendrocytes;Brain;2009-04-21
3. The neurotransmitter N-acetylaspartylglutamate in models of pain, ALS, diabetic neuropathy, CNS injury and schizophrenia;Trends in Pharmacological Sciences;2005-08-01
4. Effect of N-acetylaspartylglutamate (NAAG) on non-quantal and spontaneous quantal release of acetylcholine at the neuromuscular synapse of rat;Journal of Neurochemistry;2005-07
5. Inhibition of glutamate carboxypeptidase II (NAALADase) protects against dynorphin A-induced ischemic spinal cord injury in rats;European Journal of Pharmacology;2005-01
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