Lipoic acid improves survival in transgenic mouse models of Huntington's disease
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
General Neuroscience
Reference24 articles.
1. Interference by Huntingtin and Atrophin-1 with CBP-Mediated Transcription Leading to Cellular Toxicity
2. Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic Mice
3. Neuroprotective Effects of Creatine in a Transgenic Mouse Model of Huntington's Disease
4. Neuroprotection by the Metabolic Antioxidant α-Lipoic Acid
5. Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouse
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