Use of the Activating Gene Mutation of the Tyrosine Kinase (VAL617Phe) JAK2 as a Minimal Residual Disease Marker in Patients With Myelofibrosis and Myeloid Metaplasia After Allogeneic Stem Cell Transplantation
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference5 articles.
1. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders.;Baxter;Lancet,2005
2. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis.;Levine;Cancer Cell,2005
3. The JAK2V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic leukemia or chronic lymphoccytic leukemia.;Levine;Blood,2005
4. Mutations in innate immune system NOD2/CARD15 and TLR-4 (Thr399Ile) genes influence the risk for severe acute graft-versus-host-disease in patients who underwent an allogeneic transplantation.;Elmaagacli;Transplantation,2006
5. Real-time PCR for monitoring minimal residual disease and chimerism in patients after allogeneic transplantation.;Elmaagacli;Int J Hematol,2002
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1. Mixed myeloid chimerism and relapse of myelofibrosis after allogeneic stem cell transplantation;Haematologica;2019-07-11
2. Monitoring Minimal Residual Disease in the Myeloproliferative Neoplasms: Current Applications and Emerging Approaches;BioMed Research International;2016
3. When and how to transplant patients with myelofibrosis;Novel Insights into Myelofibrosis Pathophysiology and Treatment;2015-05
4. Evading Capture by Residual Disease Monitoring: Extramedullary Manifestation ofJAK2V617F-Positive Primary Myelofibrosis After Allogeneic Stem Cell Transplantation;Case Reports in Hematology;2015
5. Monitoring Residual Disease in the Ph-Negative Myeloproliferative Neoplasms Post-Allogeneic Stem Cell Transplantation: More Mutations and More Methodologies;Frontiers in Oncology;2014-08-08
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